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Published Online First: 10 May 2009. doi:10.1136/jcp.2009.067710
Journal of Clinical Pathology 2009;62:577-578
Copyright © 2009 by the BMJ Publishing Group Ltd & Association of Clinical Pathologists.

EDITORIAL

Swine influenza

Jonathan R Kerr

Dr Jonathan R Kerr, CFS Group, Dept of Cellular and Molecular Medicine, St George’s University of London, Cranmer Terrace, London SW17 0RE, UK; jkerr@sgul.ac.uk

Accepted 7 May 2009

The first 150 words of the full text of this article appear below.

The current epidemic of swine influenza A/H1N1, began in Mexico City in March, and has resulted in 1516 laboratory-confirmed cases, of whom 30 have died (at the time of writing, 6 May 2009). The virus has spread rapidly to 21 countries including Austria, Canada, China, Hong Kong Special Administrative Region, Costa Rica, Colombia, Denmark, El Salvador, France, Germany, Ireland, Israel, Italy, the Netherlands, New Zealand, Portugal, Republic of Korea, Spain, Switzerland and the United Kingdom. To date, all deaths have occurred in either Mexico (n = 29) or the USA (n = 1).1 By the end of April, hundreds of schools had been closed in the USA and Mexico, and the Mexican government had ordered a halt of all non-essential activities of the country for several days.2 3

Deaths have occurred predominantly in young healthy adults,4 in contrast to mortality resulting from infection with most influenza strains which result in increased . . . [Full text of this article]


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