Letter to the editorLipoprotein(a) and the acute phase response
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Serum Lp(a) concentrations are unaffected by treatment with the HMG-CoA reductase inhibitor Pravastatin: results of a 2-year investigation
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Effect of Aspirin on Lipoprotein(a) in Patients With Ischemic Stroke
2007, Journal of Stroke and Cerebrovascular DiseasesCitation Excerpt :However, most studies have reported a peak in Lp(a) levels after 8 to 10 days of onset of inflammatory conditions (i.e., longer than other acute phase reactants and a return to baseline after 4 weeks).17,18 In addition, Craig and Ledue19 did not find an acute elevation of serum Lp(a) levels similar to an acute phase response in patients with stroke. The Northern Manhattan Stroke Study showed that lipid and Lp(a) levels were not related to any change in acute phase protein and that there were no significant changes in mean serum lipids, apo-Lp, or Lp(a) levels during a 4-week period.20
Biochemical risk factors and patient's outcome: The case of lipoprotein(a)
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1996, Advances in Clinical ChemistryImpact of high lipoprotein(a) levels on in-stent restenosis and long-term clinical outcomes of angina pectoris patients undergoing percutaneous coronary intervention with drug-eluting stents in Asian population
2015, Clinical and Experimental Pharmacology and Physiology
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