The effect of LHRH and TRH on human interferon-γ production in vivo and in vitro
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Gonadotropin-releasing hormone receptor pathway affects the function of human EBV-transformed B lymphocytes in an age-independent way
2021, Experimental GerontologyCitation Excerpt :GnRH-I treatment increases the proliferation of Epstein-Barr virus (EBV)-transformed B lymphocytes and human peripheral blood mononuclear cells (PBMCs). GnRH-I treatment in stimulated PBMCs increases the expression of interleukin-2Rγ (IL-2Rγ) mRNA and synthesis of interferon γ (IFN-γ) (Chen et al., 1999; Grasso et al., 1998; Tanriverdi et al., 2005; Tanriverdi et al., 2004). The immunostimulatory effect of GnRH-I has also been suggested by the results of in vivo studies, in which GnRH-I antagonist administration in mouse models of autoimmune diseases reduced Ig levels (Jacobson et al., 1994).
Quantifying Observational Evidence for Risk of Fatal and Nonfatal Cardiovascular Disease Following Androgen Deprivation Therapy for Prostate Cancer: A Meta-analysis
2015, European UrologyCitation Excerpt :Experimental studies have shown that GnRH agonists can activate T lymphocytes and induce their proliferation. However, these effects were not observed when using GnRH antagonists [56,58]. These mechanisms could potentially explain the strong association we found between GnRH agonists and nonfatal or fatal MI and stroke.
The thyrotropin-releasing hormone (TRH)-immune system homeostatic hypothesis
2009, Pharmacology and TherapeuticsNeuroendocrine regulation of natural immunity
2005, NeuroImmune BiologyCitation Excerpt :TSHR are not detectable on foetal and neonatal immune cells. TSH significantly stimulated IL-2 and IL-12, IL-1β responses and enhanced the phagocytic activity of DCs from adult animals, enhanced the proliferative response of murine spleen cells to IL-2, significantly increased IL-2 induced NK cell cytotoxicity and enhanced the expression of MHC-II by human thyroid epithelial cells [149,152-156]. In bone marrow cells IL-6, IFNβ, TNFα, TNFβ, TGFβ2, and lymphotoxin-β responses were reproducibly induced by TSH [158].
Steroid hormones
2003, NeuroImmune Biology