Coronary artery lesions and human immunodeficiency virus infectionLésions des artères coronaires et infection par le virus de l'immunodéficience humaine
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Cited by (133)
HIV-1 Infection, Antiretroviral Therapies, and HIV-Associated Atherosclerosis
2016, Vascular Responses to PathogensCoronary artery disease and HIV; Getting to the HAART of the matter
2013, International Journal of CardiologyCitation Excerpt :However the side effects associated with the use of HAART medication introduce new concerns relating to the management of these patients. It is now recognised that HIV positive (HIV +) patients are at increased risk of developing coronary heart disease (CHD) [3–5]. This increased risk is multifactorial, with the virus itself as well as treatment with HAART and subsequent immune reconstitution considered as causative factors in the pathogenesis of atherosclerosis and coronary artery disease [6].
The patient with the human immunodeficiency virus-1 in the cardiovascular operative setting
2013, Journal of Cardiothoracic and Vascular AnesthesiaCitation Excerpt :Some studies did not show any difference from the HIV-uninfected population; however, many of these studies were of limited statistical power. In post mortem studies evaluating HIV+ patients, there has been evidence of premature coronary atherogenesis or atherosclerosis in young HIV-infected patients.91,96,97 Investigators have suggested that in the absence of risk factors for cardiovascular disease, HIV itself is the major culprit.
Comparison of in-hospital mortality from acute myocardial infarction in HIV sero-positive versus sero-negative individuals
2012, American Journal of CardiologyCitation Excerpt :Although the incidence of cardiometabolic risk and diseases has increased in seropositive patients, because of increasing life expectancy,11 some evidence suggests that variant and earlier occurring cardiovascular diseases may occur.12–14 This may in part be attributable to antiretroviral agents15–17 or unique, direct pathologic effects of HIV viremia on cardiac vasculature, including arterial endothelial inflammation, intimal fibrosis with luminal narrowing of coronary vessels, endothelial irritation, platelet dysfunction, activation of proinflammatory cytokines, thrombosis from reduced coronary blood flow, and ischemia.5,6,18 Although an increased risk for small-vessel vasculitis in HIV-infected patients has not been empirically established,19 coronary vessel disease may occur commonly in HIV-seropositive patients and increase the risk for AMI when other traditional risk factors are accounted for.
HIV-1, reactive oxygen species, and vascular complications
2012, Free Radical Biology and Medicine