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Gene expression in peripheral blood mononuclear cells from patients with chronic fatigue syndrome
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Other responses

  • Published on:
    The ubiquitous mitochondrial toxin, valinomycin, and its role in the etiology of the CFS

    Dear Drs Kaushik, Fear, Richards, McDermott, Nuwaysir, Kellam, Harrison, Wilkinson, Tyrrell, Holgate, Kerr, and Editors at JCP,

    I hope this note finds you well. Congratulations to the Physicians and Researchers who completed such admirable work regarding the possible etiology of the CFS (Chronic Fatigue Syndrome), as they made the observations that were "consistent with a complex pathogenesis involving T...

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    Conflict of Interest:
    None declared.
  • Published on:
    Rickettsial antibody levels and mitochondrial gene expression in CFS patients
    • Geoffrey C Kemp, Medical Practitioner
    • Other Contributors:
      • Robert F Townsend

    Dear Editor,

    Kaushik et al.[1] have demonstrated significantly different gene expression in a small sample of patients with chronic fatigue syndrome. They suggest these observations are consistent with a complex pathogenesis involving T cell activation and abnormalities of neuronal and mitochondrial function, possibly as a result of virus infection or organophosphate exposures.

    We suggest that chronic rick...

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    Conflict of Interest:
    None declared.