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The novel coronavirus, SARS-CoV-2, which causes COVID-19, has rapidly progressed to a global health emergency.1 COVID-19 predominantly presents with respiratory complications and associated mortality; however, emerging reports show that patients can present with cardiac involvement.1 Cardiovascular manifestations are not unique to COVID-19 as cardiac involvement have been reported with SARS-CoV and Middle East respiratory syndrome-CoV in the past.2 The pandemic in the UK has highlighted the higher risk to the Black, Asian and minority ethnic (BAME) population of adverse clinical outcomes compared with patients of white British ethnicity.3 Furthermore, BAME men aged over 50 years are noted to be at an even higher risk of severe COVID-19.4
We retrospectively reviewed the cardiac markers Troponin T, N-terminal pro B-type natriuretic peptide (NT-proBNP) and the inflammation marker C reactive protein (CRP) requested in patients with suspected COVID-19, admitted to a tertiary London hospital during the peak of the pandemic (April 2020) in the UK. Only patients who had a Troponin measurement (with or without …
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