Regular ArticleMolecular Cloning of a Novel Vascular Endothelial Growth Factor, VEGF-D
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2017, Hematology: Basic Principles and PracticePulmonary Vasculopathy Associated with FIGF Gene Mutation
2017, American Journal of PathologyCitation Excerpt :Not surprisingly, the lack of increase in VEGFR-3 signaling can be explained given the knowledge that the cysteine mutation at residue 117 results in different effects on the receptors, depending on protein concentration.19 Human FIGF is expressed in the lung before birth,23 and in adults in the heart, lung, skeletal muscle, and intestine11,20; it has recently been shown to play important roles in pulmonary vascular leak in response to hyperoxia.24 The grandfather's respiratory findings may reflect a mild disease phenotype consistent with variable expressivity or incomplete penetrance, and carriers of the gene change may show minimal, if any, phenotype; we cannot rule out as yet unidentified modifying genes or other factors to explain the disparate phenotypic findings between grandfather and grandson.
Effect of chronic hypoxia on RAGE and its soluble forms in lungs and plasma of mice
2015, Biochimica et Biophysica Acta - Molecular Basis of DiseaseCitation Excerpt :The absence of a significant difference in VEGF-B gene expression between hypoxia and normoxia exposed mice is in line with this literature [33]. VEGF-C and VEGF-D on the other hand control the growth of lymphatic vessel in lungs [34,35]. Although hypoxia has been shown not to induce VEGF-C [33] we here observed decreased VEGF-C expression.
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