Development and disease in proteinase-deficient mice: role of the plasminogen, matrix metalloproteinase and coagulation system

Thromb Res. 1998 Sep 15;91(6):255-85. doi: 10.1016/s0049-3848(98)00122-4.
No abstract available

Publication types

  • Review

MeSH terms

  • Animals
  • Arteriosclerosis / enzymology
  • Arteriosclerosis / etiology
  • Blood Coagulation / physiology*
  • Blood Coagulation Disorders / enzymology*
  • Blood Coagulation Disorders / genetics
  • Blood Coagulation Disorders / physiopathology
  • Blood Coagulation Factors / genetics
  • Blood Coagulation Factors / physiology*
  • Blood Vessels / embryology
  • Cell Movement
  • Embryonic and Fetal Development / physiology
  • Extracellular Matrix / metabolism*
  • Fetal Diseases / enzymology
  • Fetal Diseases / etiology
  • Genes, Lethal
  • Genetic Predisposition to Disease
  • Graft Survival
  • Hemorrhage / congenital
  • Hemorrhage / embryology
  • Hemorrhage / etiology
  • Hemostasis
  • Infections / enzymology
  • Inflammation / enzymology
  • Inflammation / pathology
  • Metalloendopeptidases / deficiency*
  • Metalloendopeptidases / genetics
  • Metalloendopeptidases / physiology
  • Mice
  • Mice, Inbred Strains
  • Mice, Knockout
  • Morphogenesis / physiology
  • Myocardial Ischemia / enzymology
  • Myocardial Ischemia / etiology
  • Neoplasms / enzymology
  • Neoplasms / pathology
  • Neovascularization, Physiologic
  • Plasminogen / deficiency*
  • Plasminogen / genetics
  • Plasminogen / physiology

Substances

  • Blood Coagulation Factors
  • Plasminogen
  • Metalloendopeptidases